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Archive for August, 2013

Grid Diary

Grid Diary – The simplest way to get started with keeping a diary (3.5 stars with 126 User Ratings) 
iPhone App 
$2.99 → Free 

 

 

griddiary

Grid Diary is a simple diary with a very easy to navigate interface that offers a unique way to encourage you to write. The diary will ask you questions that give your memory a good kick in the behind to get started on your writing. This gives you that extra bit of edge in recording the best moment of your day. Grid Diary can also motivate you to complete goals you set by asking questions such as what goals have you accomplished today or set three small goals for tomorrow. Once you’ve written some entries, you can search through your past entries via a keyword search functionality, and see everything laid out on a calendar grid so you can jump to specific days. If you want to keep spying eyes out, Grid Diary also features a passcode function. Whether you like writing a diary or not, Grid Diary is worth seeing if it can help.

via Best Free Apps of the Day on 8/29. Photo Manager Pro, iClean, Grid Diary, & More! | App Chronicles.

Black-Bottom Peanut Butter Mousse Pie

black-bottom-peanut-butter-mousse-pie
INGREDIENTS

Nonstick vegetable oil spray

7 whole graham crackers, coarsely broken

1/4 cup (1/2 stick) unsalted butter, melted

4 tablespoons sugar, divided

1 1/3 cups bittersweet or semisweet chocolate chips (about 8 ounces)

2/3 cup plus 1 3/4 cups chilled whipping cream, divided

2 tablespoons light corn syrup

2 teaspoons vanilla extract, divided

6 ounces (1 cup) peanut butter chips

2 tablespoons creamy peanut butter (do not use old-fashioned style or freshly ground)

PREPARATION

 

  • Preheat oven to 350°F. Spray 9-inch-diameter glass pie dish with nonstick spray. Blend graham crackers, melted butter, and 2 tablespoons sugar in processor until moist clumps form. Press crumb mixture over bottom and up sides of prepared pie dish. Bake crust until lightly browned, about 15 minutes.
  • Meanwhile, combine chocolate chips, 2/3 cup cream, corn syrup, and 1 teaspoon vanilla in microwave-safe bowl. Microwave on medium heat until chocolate softens, about 3 minutes. Whisk until melted and smooth. Spread chocolate mixture over bottom of crust. Freeze 10 minutes.
  • Microwave peanut butter chips and 3/4 cup cream in large microwave-safe bowl on medium heat at 15-second intervals just until chips soften, stirring often. Whisk in peanut butter and 1 teaspoon vanilla. Cool to barely lukewarm. Beat remaining 1 cup cream and 2 tablespoons sugar in medium bowl until very thick but not yet holding peaks; fold into peanut butter mixture in 3 additions. Spoon mousse over chocolate layer. Chill at least 1 hour and up to 1 day.

via Black-Bottom Peanut Butter Mousse Pie – Bon Appétit.

Malaria researchers propose targeting breeding sites

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Mosquitoes are increasingly resistant to insecticides

 

A new report says targeting mosquito breeding sites is likely to be increasingly necessary to reduce cases of malaria in Africa and Asia.

Researchers say that with mosquitoes becoming ever more resistant to insecticides, new approaches will be needed to help control the disease.

They include flushing out stagnant water where mosquito larvae grow and treating water with chemicals.

More than 600,000 people died from the malaria in 2010, most African children.

The number of deaths from malaria has fallen by a quarter in the last decade, largely thanks to the widespread distribution of mosquito nets treated with insecticides and the use of indoor insecticides sprays.

But the insects are becoming increasingly resistant to these chemicals, so a new report by researchers at the London School of Hygiene and Tropical Medicine says authorities should also use a method called “larval source management”.

This is where mosquito larvae found in stagnant water like paddy fields or ditches are killed off by draining or flushing the land before they get a chance to develop. It also involves something called larviciding where chemicals are added to standing water.

The study found evidence that the method may significantly reduce both the number of cases of malaria by up to 75% and the proportion of people infected with the malaria parasite by up to 90% when used in appropriate settings.

The report’s authors trawled through thousands of studies looking at the effectiveness of this method and found 13 which reached a high enough standard to draw their conclusions. The research came from countries including The Gambia, Kenya, Mali and the Philippines.

The report’s author Lucy Tusting says the findings have important implications for malaria control policy

“The tremendous progress made in malaria control in the last decade is now threatened by mosquito resistance to the insecticides available for long-lasting insecticide treated nets and indoor residual spraying.” she says

“Thus additional methods are needed to target malaria-transmitting mosquitoes.

Our research shows that larval source management could be an effective supplementary intervention in some places.”

The World Health Organization says the research is not robust enough to support this method, and it is not recommended for use in rural areas where breeding grounds are hard to find.

A WHO spokesperson said: “Until there is more compelling evidence, larval control should continue to be viewed as a supplementary measure for malaria control in carefully selected settings. Promoting the widespread use of larval source management in rural areas of sub-Saharan Africa would be premature.”

The WHO says larval source management should only be used alongside insecticide sprays and nets.

via BBC News – Malaria researchers propose targeting breeding sites.

Bullied mice overcome anxiety after light treatment

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Neurons were found (green) which inhibited the release of serotonin (red)

Scientists have discovered the neurons that can lead mice to become socially anxious when they are bullied.

These neurons inhibit the release of serotonin, the deficiency of which is linked to social disorders.

The team was able to “switch off” these neurons which made the mice resilient to bullying.

The work is published in the Journal of Neuroscience, and could give insight into how similar structures in humans function.

Olivier Berton at the University of Pennsylvania medical school, US, said his paper provided a novel understanding at the cellular level, of how social defensiveness developed in a mouse.


How anxiety affects social cues

  • Our brains have evolved to pick up important social cues from others, which in turn helps us anticipate threat and to avoid potentially undesirable interactions
  • But when social affective disorders such as anxiety and depression are present, these appraisal systems no longer function as they should
  • Previous studies have shown that decreasing levels of serotonin in the brain leads both healthy and those with social deficits to perceive neutral faces to be more hostile. When given anti-depressants this is reversed
  • There are numerous areas of the brain that are known to relate to social disorders but at the cellular level there remains much to be discovered, which this new work aims to do

Source: Olivier Berton and colleagues

Dr Berton and colleagues exposed 54 mice to negative social experiences from aggressive, territorial mice and found that more than half were defeated by bullies, which made them anxious.

The neurons responsible for emitting serotonin consequently became less responsive.

Resilient mice

The team identified that a class of GABA neurons (gamma aminobutyric acid – the neurotransmitters which inhibit other cells) were next to the neurons which released serotonin.

It was these GABA neurons that were putting a “brake” on the serotonin being released.

Using optogenetics, a technique which can make individual neurons respond to light, the team was able to switch off these neighbouring GABA neurons which made the mice resilient to bullying.

After the GABA neurons had been deactivated, it changed the way mice perceived a social threat and therefore prevented them becoming socially anxious.

“We are removing the brake from serotonin neurons and they are then able to fire normally,” explained Dr Berton.

A mouse receiving inhibition while in sensory contact with an aggressor
The team was able to switch off neurons using a technique called optogenetics

Finding the function of these GABA neurons could now help scientists understand why current antidepressants do not work for everyone and targeting these neurons could improve medication, he added.

“It could help us understand the basis of similar social symptoms in human pathologies, like social phobias and depression.”

Dr Berton explained that while there has been much progress made indentifying regions of the brain involved in social disorders, more needs to be done to learn about the cellular level of the brain.

“This work may give us a stepping stone to extrapolate about how this is happening in the human brain,” he told BBC News.

Lynn Kerby of the Temple University School of Medicine, Philadephia, US, was not involved with the research.

She said the study could give “significant insight” into the behaviours that contributed to social stress in humans.

“If there was a way to control these circuits in the human brain with the cellular precision used in the mice in this study, the results suggest that we might be able to promote adaptive behavioural responses to stress.

“Though exciting to imagine, whether such manipulations will ever be available or practical for use to treat human psychiatric diseases in the future, remains to be seen.”

via BBC News – Bullied mice overcome anxiety after light treatment.

Protein clue to old-age memory loss

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Why does memory decline in old age?

A clue to why memory deteriorates with age has been found by US researchers.

Experiments on mice suggested low levels of a protein in the brain may be responsible for memory loss.

It is hoped the discovery could lead to treatments to reverse forgetfulness, but it is a big leap from the mouse to a human brain.

The study, published in the journal Science Translational Medicine, said age-related memory loss was a separate condition to Alzheimer’s disease.

The team at Columbia University Medical Centre started by analysing the brains of eight dead people, aged between 22 and 88, who had donated their organ for medical research.

They found 17 genes whose activity level differed with age. One contained instructions for making a protein called RbAp48, which became less active with time.

Memory boost

Young mice genetically engineered to have low RbAp48 levels performed as poorly as much older mice in memory tests.

Using a virus to boost RbAp48 in older mice appeared to reverse the decline and boosted their memory.

One of the researchers, Prof Eric Kandel, said: “The fact that we were able to reverse age-related memory loss in mice is very encouraging.

“At the very least, it shows that this protein is a major factor, and it speaks to the fact that age-related memory loss is due to a functional change in neurons of some sort. Unlike with Alzheimer’s, there is no significant loss of neurons.”

It is still not know what impacted adjusting levels of RbAp48 in the far more complex human brain or even it if is possible to manipulate levels safely.

Dr Simon Ridley, from Alzheimer’s Research UK, said: “While the findings may seem clear cut from these studies, in reality people reaching older age may well have a combination of changes happening in the brain – both age-related and those involved in the early stages of Alzheimer’s.

“Separating early changes in Alzheimer’s from age-related memory decline in the clinic still presents a challenge, but understanding more about the mechanisms of each process will drive progress in this area.”

via BBC News – Protein clue to old-age memory loss.

Miniature ‘human brain’ grown in lab

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Cross-section of miniature human brains termed cerebral organoids

Miniature “human brains” have been grown in a lab in a feat scientists hope will transform the understanding of neurological disorders.

The pea-sized structures reached the same level of development as in a nine-week-old foetus, but are incapable of thought.

The study, published in the journal Nature, has already been used to gain insight into rare diseases.

Neuroscientists have described the findings as astounding and fascinating.

The human brain is one of the most complicated structures in the universe.

Scientists at Institute of Molecular Biotechnology of the Austrian Academy of Sciences have now reproduced some of the earliest stages of the organ’s development in the laboratory.

Brain bath

They used either embryonic stem cells or adult skin cells to produce the part of an embryo that develops into the brain and spinal cord – the neuroectoderm.

This was placed in tiny droplets of gel to give a scaffold for the tissue to grow and was placed into a spinning bioreactor, a nutrient bath that supplies nutrients and oxygen.

Brain
A cerebral organoid – the brown pigments are a developing retina

 

The cells were able to grow and organise themselves into separate regions of the brain, such as the cerebral cortex, the retina, and, rarely, an early hippocampus, which would be heavily involved in memory in a fully developed adult brain.

The researchers are confident that this closely, but far from perfectly, matches brain development in a foetus until the nine week stage.

The tissues reached their maximum size, about 4mm (0.1in), after two months.

The “mini-brains” have survived for nearly a year, but did not grow any larger. There is no blood supply, just brain tissue, so nutrients and oxygen cannot penetrate into the middle of the brain-like structure.

One of the researchers, Dr Juergen Knoblich, said: “What our organoids are good for is to model development of the brain and to study anything that causes a defect in development.

“Ultimately we would like to move towards more common disorders like schizophrenia or autism. They typically manifest themselves only in adults, but it has been shown that the underlying defects occur during the development of the brain.”

The technique could also be used to replace mice and rats in drug research as new treatments could be tested on actual brain tissue.


I think it’s just mindboggling”

Prof Paul MatthewsImperial College London

‘Mindboggling’

Researchers have been able to produce brain cells in the laboratory before, but this is the closest any group has come to building a human brain.

The breakthrough has excited the field.

Prof Paul Matthews, from Imperial College London, told the BBC: “I think it’s just mindboggling. The idea that we can take a cell from a skin and turn it into, even though it’s only the size of a pea, is starting to look like a brain and starting to show some of the behaviours of a tiny brain, I think is just extraordinary.

“Now it’s not thinking, it’s not communicating between the areas in the way our brains do, but it gives us a real start and this is going to be the kind of tool that helps us understand many of the major developmental brain disorders.”

The team has already used the breakthrough to investigate a disease called microcephaly. People with the disease develop much smaller brains.

Brain with microcephaly
A much smaller brain develops with microcephaly

By creating a “mini-brain” from skin cells of a patient with this condition, the team were able to study how development changed.


It’s a long way from conscience or awareness or responding to the outside world. There’s always the spectre of what the future might hold, but this is primitive territory”

Dr Zameel CaderJohn Radcliffe Hospital

They showed that the cells were too keen to become neurons by specialising too early. It meant the cells in the early brain did not bulk up to a high enough number before specialising, which affected the final size of even the pea-sized “mini-brains”.

The team in Vienna do not believe there are any ethical issues at this stage, but Dr Knoblich said he did not want to see much larger brains being developed as that would be “undesirable”.

Dr Zameel Cader, a consultant neurologist at the John Radcliffe Hospital in Oxford, said he did not see ethical issues arising from the research so far.

He told the BBC: “It’s a long way from conscience or awareness or responding to the outside world. There’s always the spectre of what the future might hold, but this is primitive territory.”

The “mini brain” is roughly the size and developmental level of a nine-week foetus

Dr Martin Coath, from the cognition institute at Plymouth University, said: “Any technique that gives us ‘something like a brain’ that we can modify, work on, and watch as it develops, just has to be exciting.

“If the authors are right – that their ‘brain in a bottle’ develops in ways that mimic human brain development – then the potential for studying developmental diseases is clear. But the applicability to other types of disease is not so clear – but it has potential.

“Testing drugs is, also, much more problematic. Most drugs that affect the brain act on things like mood, perception, control of your body, pain, and a whole bunch of other things. This brain-like-tissue has no trouble with any of these things yet.”

via BBC News – Miniature ‘human brain’ grown in lab.

檳城‧蚊症病例飆升18區亮紅燈‧一起防毒蚊

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阿菲夫說,相比去年同期,今年的骨痛熱症病例狂飆。(圖:星洲日報)

年初至今已有482宗病例,2人死亡,上週25人患病;州內18區亮紅燈,有關地區的居民受促提高警惕。

檳州衛生事務委員會主席阿菲夫昨日(28日)在檳州首席部長林冠英及其他行政議員的陪同下召開新聞發佈會。

阿菲夫說,相比去年同期,今年的骨痛熱症病例狂飆,不幸患病死亡的人是一名來自峇央峇魯區的1歲嬰孩及一名來自喬治市的79歲年長者,死亡案例發生在今年6月。

阿菲夫:人口移動提高傳染率
開齋節後病例突增

阿菲夫說,病例在開齋節後突然增加,今年第三十三週,共有18宗病例,第三十四週則有25宗病例。

他認為,這是由於開齋節時有人口大量移動,而增高了傳染率,佳節時期也缺乏清潔活動。

他說,這一週的骨痛熱症熱點是西南區的斯里德里瑪組屋(Flat Sri Delima)和東北區的雙溪檳榔第一條路(Lebuh Sungai Pinang1)。檳州衛生局也圈下了另18個骨痛熱症熱點,這地區的居民在未來一週內需格外小心,若身體出現骨痛熱症狀,就需馬上到醫院檢血斷定。

拉昔:每月至少一次
各區鄉委會須辦大掃除

檳州第一副首長拉昔說,鄉委會事務由他掌管,他將下令各區共291個鄉委會每個月至少要舉辦一次大型清潔活動。

他說,鄉委會需關注此事,並與檳州衛生局緊密合作,帶動社區居民一起對抗骨痛熱症;其中一個方式就是在住宅區進行清潔活動,消滅黑斑蚊的溫床(積水處)。

阿菲夫表示,檳州衛生局已準備了滅蚊藥,公眾可以自行領取,該局也將在黑區噴蚊霧。

即起徵免費乳房檢查計劃書

阿菲夫說,州政府即日起向私人醫院和診所徵求“免費乳房檢查”計劃書,9月30日截止。

他說,州政府將提供“免費乳房檢查”給州內女性,並準備提供交通工具載送女性前往接受檢查。

州政府即日起向私人醫院和診所徵求“免費乳房檢查”計劃書,有意者可瀏覽州政府官方網站,取得更多詳情。

 


 

你知道嗎? 

骨痛熱症簡介
黑斑蚊傳染病毒

骨痛病毒只能存於人、猴子及病媒蚊(黑斑蚊)體內,病毒則藉由黑斑蚊叮呅才能傳給人。

骨痛熱症在病患開始發燒的前一天,直到退燒期間,都具有傳染力。病毒在黑斑蚊體內經過8至12天的成長後,便可以傳給他人,期間可能長達數個月,依此循環,直到這隻蚊子死亡。

症狀包括發燒(39度至40度)或惡寒、皮膚出疹、四肢酸痛、肌肉痛、前額頭痛、後眼窩痛、腹部痛、背痛、喉痛、嘔吐、頭暈、牙齦出血、吐血或血便、可能出現疲倦及抑鬱症狀。

via 2013年08月29日 – 檳城‧蚊症病例飆升18區亮紅燈‧一起防毒蚊 – 大北馬 – 地方.

孩子不想吃 10撇步矯正偏食

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國外研究發現,和父母共餐的孩子會多吃蔬果,不容易有偏食習慣。

 

國內幼童偏食情況越來越嚴重,甚至不愛吃蔬果。營養師表示,偏食將影響孩子的腦力與身體肌肉發展,更會影響學習表現;國外研究發現,和父母共餐的孩子會多吃蔬果,不容易養成偏食習慣;建議家長只要在餐點上多用心,以身作則一起用餐,就能愈早改善孩子偏食的情況。

英國利茲大學研究發現,總是和家人一起用餐的兒童每天會多吃125克的蔬果,偶爾和家人一起用餐的兒童每天也會多吃95克的蔬果;孩子長時間待在家中和學校,老師和家長的陪伴和帶領,有非常大的影響和示範作用。

董氏基金會營養師尤宣文表示,其實孩子的口味喜好,並非天生,正確的飲食行為需要從小培養,父母只要把握關鍵時間,讓孩子從小多接觸全穀、蔬果、牛奶等健康食物,運用好看的顏色、擺盤和餐具擺設,自然可以引起孩子的興趣。

蔬果顏色 誘發對食物興趣

例如,小孩子偏愛酸甜的口感,家長可以利用蔬果五彩繽紛的顏色、改變烹調方式,像是切丁、作成動物造型引起孩子興趣,在絞肉中增加蔬菜、蘿蔔泥,也可以讓孩子逐漸習慣味道,或者是讓小朋友自己動手,有參與感,也比較願意接受蔬菜。

建議家長,只要多看多聽多試,孩子經過幾周接觸、試吃不同的蔬果後,食物接受度都能大幅提升,甚至家長可以試試「每天挑戰一種新食物」的遊戲,要求孩子嘗試原本沒吃過,或是不敢吃的苦瓜、洋蔥、秋葵、龍鬚菜、茄子等食物。

10個小秘訣 讓孩子不挑食

祕訣1:多看多聽多試

營養師:讓孩子從小多接觸各種食物,藉由熟悉各種食物的顏色及形狀,解說內含營養對健康的好處。

祕訣2:多鼓勵不強迫

營養師:讓孩子自行選擇自己喜歡的健康食物,再搭配一兩樣新食材,鼓勵孩子嚐試並自行決定吃多少及剩多少。

祕訣3:好看更好吃

營養師:運用好看的顏色、擺盤和餐具擺設,引起孩子的興趣。

秘訣4:好心情好胃口

營養師:不在餐桌上訓話,多與孩子聊聊趣事,心情開朗,食物也會變好吃。

秘訣5:參與改變一切

營養師:與孩子一同準備餐食,不但能讓孩子有成就感,還懂得珍惜食物。

秘訣6:食物變身秀

營養師:換個方式煮出美味,變化烹調方式保持食物新鮮感,訓練孩子的多元口味。

秘訣7:想像力量大

營養師:用劇情引導孩子嚐試食物,玩出好食慾的同時,還能夠激發想像力。

秘訣8:明訂遊戲規則

營養師:別把零食當獎勵,訂下規則讓孩子知道零食只能偶爾吃,需在正餐之後吃。

秘訣9:健康吃快樂動

營養師:運動後享用食物,美味加倍。

秘訣10:一起吃最好吃

營養師:養成全家一起在家用餐共識,從小養成孩子在餐桌上用餐的習慣,美食親情兼具的餐桌,會讓「好吃的記憶」留在孩子心中。

via 孩子不想吃 10撇步矯正偏食 | 20130829 | 華人健康網.

慢速肌肉鍛鍊法 不讓肌肉變脂肪

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肌肉長時間不鍛鍊的話,就有可能造成脂肪細胞變多。(圖片提供/美忍者)

學生時期怎麼樣都不會變胖,但是出社會後身體卻像氣球一樣一天比一天脹! 你如果也是這樣就要注意了!一直推託因為工作忙碌而沒有時間運動的人,身體將會在不知不覺中發生超出你預想的情況唷!

大家應該都有聽說過一旦停止運動的話,原有的肌肉會轉變成脂肪這個說法吧? 告訴大家,別不相信,因為這是真的!這回要來向大家介紹肌肉轉變成脂肪的3個因素以及對策。

1:因為原本發達的肌肉衰退,而使脂肪有趁隙而入的機會

原本發達的肌肉,不繼續維持其強度,一旦衰退後會在肌肉細胞之間產生讓脂肪進入的空間。進入這個空間的脂肪被稱作筋細胞外脂肪。筋細胞外脂肪是會在肌肉之間漸漸增多的特殊脂肪。大家應該有看過霜降肉吧?就大概是像那個樣子!這種脂肪一旦增多,肌肉就會無法支撐其重量而導致下垂。

2:產生肌肉的「基礎」刺激不足

運動時給予肌肉刺激的話、就會產生新的肌肉。在發展成肌肉時的基礎稱之為、“筋衛星細胞”。這種細胞在尚未成熟的狀態下、會分泌讓肌肉伸縮成長的荷爾蒙、使肌肉變得發達。可是,一旦運動的刺激變少,成長荷爾蒙的分泌也會變少。而筋衛星細胞竟有可能會轉變成恐怖的脂肪細胞。

3:肌肉的營養不良

偏激的飲食減肥會有導致肌肉變成霜降肉的可能性。特別是製造肌肉的材料蛋白質不足的話、肌肉會漸漸變細,成長荷爾蒙的分泌也會減少、大大提高肌肉霜降化的可能性。

大家知道了以上3個原因之後,那到底要如何才能不長脂肪呢?

在NHK的節目『實驗大百科』的實驗當中、得知了慢速肌肉鍛鍊運動是最有效的。慢速肌肉鍛鍊運動按照字面就是緩慢地進行鍛鍊。就是因為緩慢所以負擔會比較小、但是卻會源源不絕地讓肌肉受到壓力,持續讓肌肉內的血管受到壓迫,這樣的方式和進行激烈的訓練是一樣的。這個時候的腦會誤以為身體在進行激烈運動,而分泌出許多的荷爾蒙。

因為負擔小,所以比較不會有肌肉痠痛的問題。對於其它部位的負擔較小,但是卻可以分泌出成長荷爾蒙,應該是大家想立刻想嘗試的方法吧?若不想脂肪鬼上身,就趁肌肉尚未衰退之前持續身體的鍛練吧!讓自己快快擺脫霜降脂肪,擁抱輕盈的人生!

via 慢速肌肉鍛鍊法 不讓肌肉變脂肪 | 20130829 | 華人健康網.

高溫破表!防皮膚烤焦先擦防曬乳

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避免被曬傷,最重要的是做好防曬萬全準備。除了遮陽用品派上用場之外,防曬乳更是不能離手。

雖然時序已經進入立秋,但是近來天氣真的熱翻天啦!台北市一度創下117年來最高溫紀錄,出現攝氏39.3度高溫,許多人走在路上熱到爆,甚至感覺皮膚快「烤焦」了,全身發紅,熱烘烘的,還忍不住開始抓癢。其實,這代表「你已經曬傷了」,如果能事前做好抗陽大作戰,就可以遠離曬傷的威脅了。

容易被曬傷的部位包括身上較突出的部份如臉頰、鼻子、頸背、耳朵,或是平常上班時被衣服遮住的肩膀、腿,都可能是曬傷最嚴重的部位。避免被曬傷,就要禁得起烈陽的「烤」驗,最重要的是做好防曬萬全準備。除了遮陽用品派上用場之外,防曬乳更是不能離手,出門前使用還不夠,如果長時間在戶外活動,更需要適時的補充。

有人曬傷後,竟然使用偏方,例如塗抹白花油、萬金油、薄荷油、蘆薈霜等產品,結果不但未緩解皮膚發熱感覺,甚至更加有燒灼感。這一些用品有的可以保濕,但是無助於緩解曬傷傷勢,無法消散皮膚表面熱度。加上屬於油膏類產品,在已經曬傷的皮膚表皮,反而增加另一種「負荷」,使得傷勢愈演愈烈。

坊間防曬乳百百款,怎麼選才正確安全呢?防曬產品必需選擇溫和不含香精、刺激性成分,並且能抗UVA防止光老化與抗UVB防曬傷,都是基本必備條件,在防曬係數選擇上最好採用效係數SPF50的產品。使用方式為出門前30分鐘擦防曬乳,每隔2~3小時補充1次。油性肌膚、青春痘膚質或從事一般日常生活工作的人,需選擇清爽且不產生粉刺的防曬品(親水性的防曬乳);若為乾性皮膚,又要去海邊外島度假,就必須選擇親脂性、不易脫落又保濕的防曬霜出門。

 

油性肌膚、青春痘膚質,需選擇清爽且不產生粉刺的防曬品(親水性的防曬乳)。(攝影/楊伯康)

油性肌膚、青春痘膚質,需選擇清爽且不產生粉刺的防曬品(親水性的防曬乳)。(攝影/楊伯康)

 

到底該如何選擇安全有效的防曬用品呢?全球10個皮膚癌患者中就有8個來自於澳洲,而澳洲也是全球罹患皮膚癌最嚴重的國家,因此,能通過澳洲藥物管理局認證的防曬產品,在品質上有保障,且防曬效果較佳。尤其防曬產品通過人體(in vivo)實驗,證實能真正有效隔離紫外線中的UVA、UVB的防曬產品,是不錯的選擇,小孩與大人皆適用。

【防曬ABC原則】:

★1.A(Avoid):遠離強烈陽光在上午10時到下午2時為陽光最強烈時刻,建議少出門。

★2.B(Block):使用防曬品遮蔽,若要暴露在陽光下,包括登山,或到海邊、室外游泳池戲水,要利用防曬產品阻隔紫外線。

★3.C(Cover):減少肌膚曝露在紫外線的面積,外出時盡可能撐傘、戴寬邊帽子、穿長袖衣服、戴上太陽眼鏡,以及擦防曬乳。

via 高溫破表!防皮膚烤焦先擦防曬乳 | 20130827 | 華人健康網.

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